Results: About one-third of the observed prevalence decline through 2010 can be attributed solely to fewer parents smoking after the initial education shock. Combining peer-group cessation contagion explains well over one-half of the total historical prevalence reduction. Plausible additional echo effects could explain the entire historical reduction in smoking prevalence.
Conclusions: Ongoing anti-smoking interventions are credited with ongoing reductions in smoking, but most, or perhaps all that credit really belongs to the initial education and its continuing echoes. Ensuring that people understand the health risks of smoking causes large and ongoing reductions. The effect of all other interventions (other than introducing appealing substitutes) is clearly modest, and quite possibly, approximately zero, after accounting for the echo effects.
We know that choosing to smoke is socially contagious – the more people around someone who smokes, particularly their parents, the more likely they are to start smoking.1 Parental smoking is the most consistent strong predictor of whether a teenager (of a particular age, in a particular population) will start smoking. Smoking prevalence among siblings, peer groups, and the wider community affects uptake via overt and subconscious social signaling. All of these are taken as fact in the scientific literature and in Phillips et al tobacco control politics, where they are cited as motivation or points of leverage for interventions. But one important implication – that a downward shock or trend in smoking prevalence will, by itself, cause further downward trending for more than a generation – is generally ignored.
Similarly, smoking cessation is a contagious behavior. This is particularly clear for switching to a lower-risk alternative, wherein the person quitting smoking demonstrates to their social contacts that the choice is appealing and educates them about the alternative. However, even if the choice of cessation method is not affected by social-contact education, the demonstration effect of quitting itself is still powerful. Seeing a friend quit smoking takes it from being an abstract possibility to a concrete example of success. In addition, simply having fewer people who smoke in one’s social circles encourages quitting. Each of these, and all of them together, creates a positive feed-forward effect from any smoking reduction.
Thus, a one-time permanent downward shock in the popularity of smoking – like that caused by initial education about the harms from smoking – causes a long tail of transition to a new lower equilibrium, echoes of the initial shock. If many people quit smoking, then many more who would have started smoking had they come of age earlier will not do so and others will be motivated to quit over time. The subsequent cohorts coming of age not only will experience the effect of the downward shock, but also be subject to less social contagion. There will be a new equilibrium, but it will only be reached slowly, with a substantial portion of the effect taking more than a generation. This will happen with or without any further efforts to discourage smoking. Subsequent interventions could still have effects beyond the secular trend toward a new equilibrium, of course, but it makes no sense to try to quantify those effects without trying to estimate the background effects of the echoes alone.
That would certainly help explain why tobacco-style regulation fails to work when applied to other activities. These policies tend to focus on the Three As - affordability, advertising and availability - but whilst it is Econ 101 to note that higher prices tend to lead to lower consumption, albeit at the expense of consumers, the evidence for the other two As is remarkably thin on the ground.
Take alcohol. A 2019 systematic review titled 'Do alcohol control policies work?' and written by two members of the South African Medical Research Council concluded that ‘[r]obust and well-reported research synthesis is deficient in the alcohol control field despite the availability of clear methodological guidance.’ The policies examined included restricting alcohol advertising and restricting on- and off-premise outlet density.
With regards to advertising, a Cochrane Review, which is usually considered definitive, found 'a lack of robust evidence for or against recommending the implementation of alcohol advertising restrictions'.
Even the authors of Alcohol: No Ordinary Commodity, the bible of the secular temperance movement, were only able to make a limp case for advertising bans.
‘Imposing total or partial bans on advertising produce, at best, small effects in the short term on overall consumption in a population, in part because producers and sellers can simply transfer their promotional spending into allowed marketing approaches. The more comprehensive restrictions on exposure (e.g. in France) have not been evaluated… The extent to which effective restrictions would reduce consumption and related harm in younger age groups remains an open question.’
A systematic review published in 2012 tried very hard to find evidence to support orthodox, supply-side anti-alcohol policies. It was written by dyed-in-the-wool 'public health' activists, including Mark Petticrew and Martin McKee, but they really struggled to find what they wanted.
On advertising, they found seven studies which 'provided inconclusive results for the influence of advertising on alcohol use'.
There wasn't much evidence and a lot of it was of poor quality, but...
A study rated as ‘strong’ in the quality assessment found no significant association between exterior advertising in areas near schools and adolescent drinking.
In general, the findings of this review are consistent with reviews on wider alcohol availability (Popova et al., 2009), which have found that availability has a strong influence on alcohol use.
In general, the results of this review are similar to those found in previous reviews (Babor et al., 2003)—studies show mixed results but strongly indicate that greater exposure to advertising is associated with higher levels of alcohol use.
They also looked at availability - including licensing hours and outlet density - and again struggled to find evidence to support their priors. They found '21 studies on the influence of availability of alcohol from commercial sources on alcohol use', but, alas...
Overall the findings provided inconclusive results for the influence of availability on alcohol use, although some studies indicated that higher outlet density in a community may be associated with an increase in alcohol use.
With regards outlet density specifically:
For off-premise outlets (such as shops), eight studies found no significant association but there is some indication that a higher density of off-premise outlets may be associated with an increased likelihood of heavy drinking. For on-premise outlets (such as bars and restaurants), results were also mixed but there is some indication that a higher density of on-premise outlets may be associated with an increase in the likelihood of drinking and heavy drinking.
As for local changes to licensing regulations...
Four studies (with four effect estimates) looked at the influence of local licensing changes on alcohol use, which included banning alcohol sales and making changes to the hours, days and volumes of alcohol sales that were licensed. They indicate that licensing restrictions may reduce alcohol use, but the evidence is not very robust.
Consumption of sugar-reduced products, as part of a blinded dietary exchange for an 8-week period, resulted in a significant reduction in sugar intake. Body weight did not change significantly, which we propose was due to energy compensation.
This week saw the publication of a systematic review of food advertising. Again, it was written by fervent interventionists and its lead author is the activist-academic Emma Boyland who is responsible for a fair chunk of the literature herself. She is now not only a professor but also an advisor to thee World Health Organisation.
Evidence on diet and product change was very limited. The certainty of evidence was very low for four outcomes (exposure, power, dietary intake, and product change) and low for two (purchasing and unintended consequences).
Their research, published in JAMA Pediatrics, found that food marketing was associated with significant increases in food intake, choice, preference, and purchase requests. However, there was no clear evidence of relationships with purchasing, and little evidence on dental health or body weight outcomes.
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